Aging (or stage) of ischemic stroke is important in a number of clinical settings. Both CT and MRI can help in determining when a stroke occurred. Based on multiple sequences, especially DWI and ADC, MRI has not o...
This specific ischemic change on MRI histologically corresponded to selective neuronal death and gliosis with preservation of the macroscopic structure of the brain. A similar MRI pattern reported in patients who have sustained brief ischemia ...
The relationship between cerebral ischemia and glioma is still ambiguous based on molecular mechanisms, but several clinical reports and case studies have indicated that glioma and cerebral ischemia can facilitate each other with respect to occurrence. It has been reported that the location of the tumo...
or oval and positioned within the vessel wall along the axis of the capillary, while the pericyte nucleus is spherical, small but stain densely, and generally have a bulgy position on the capillary wall. The intact retinal wholemounts with high-quality staining were coded and subsequently used f...
CONCLUSIONS: Brief MCAO with reperfusion induces the delayed ischemic changes of hyperintensity and hypointensity on T1W and T2W MRI, respectively, in the rat striatum with high reproducibility. This specific ischemic change on MRI histologically corresponded to selective neuronal death and gliosis ...
semiovale and (iii) deep prominent sulci, abutting the ventricle. These findings can be identified on both T1 and T2 weighted imaging. MRI has the advantage that an increased signal intensity on T2 weighted imaging highlights areas of presumed gliosis. Once myelination has occurred, the fluid ...
protein-1, IL-1β [40], glial fibrillary acidic protein (GFAP), vimentin, and nestin that can lead to reactive gliosis and scar formation [41]. After stroke, due to failure of Na+, K+pump, astrocytes swell, which leads to high intracerebral pressure and less cerebral perfusion [42]....
In contrast, late seizures are related to neuronal circuit reorganization by aberrant gliosis and development of hyperexcitable cicatrix, eventually resulting in spontaneous seizures [23, 36]. We hypothesized that clinical characteristics of PSSi patients might reflect the epileptogenic predisposition of ...
The remainder corresponded to preexisting gliosis, cerebrospinal fluid, or was lost to postinfarct atrophy. The apparent diffusion coefficient of core was lower than that of noncore voxels (P −6 mm2/s (sensitivity 69% and specificity 78%).ConclusionsOur data suggest that the ischemic ...
ischemic brain injury suggest the commonest lesion is wide- spread WM gliosis (3,6). Placental abruption is an identified antecedent factor (7). Regional differences in vulnerability to HI exist (8,9). Pre- term WM is susceptible to HI, and inflammation due to the selective vulnerability ...