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Overall, MRI classes reflected typical histopathological FCD characteristics: Class-1 was associated with severe WM gliosis and interface blurring, Class-2 with severe GM dyslamination and moderate WM gliosis, Class-3 with moderate GM gliosis, Class-4 with mild interface blurring. A detection ...
177 Fibrin-selectivity is important since the agent tends to bind at the site of the thrombus and not cause systemic fibrinogenolysis. In DIAS, patients were selected for fibrinolysis if they had a diffusion/perfusion mismatch on MRI and were treated within a 3- to 9-hour window. The ...
(n ¼ 54), periventricular white matter alterations (n ¼ 40), contusions (n ¼ 22), hamartia (n ¼ 10), calcifications (n ¼ 7), vascular sclerosis (n ¼ 1), subependymal gliosis (n ¼ 1), perivascular lymphocytes (n ¼ 1), infarct (n ¼ 1), and neuronal ...
MRI protocol Data were acquired on a 3T MRI scanner (TIM Trio, Siemens Medical Solution, Erlangen, Germany) equipped with an eight-channel head coil. We used a 3D rapid gradient echo sequence to acquire high-resolution T1-weighted MRI (T1w; 176 slices; repetition time (TR) = 2300 ...
CONCLUSIONS: Brief MCAO with reperfusion induces the delayed ischemic changes of hyperintensity and hypointensity on T1W and T2W MRI, respectively, in the rat striatum with high reproducibility. This specific ischemic change on MRI histologically corresponded to selective neuronal death and gliosis ...
Thirteen of those 16 (81%) of the 17 remote cortical regions that were covered by subdural EEG were around cortex showing rapid seizure spread on intracranial EEG. Remote FMZ PET abnormalities were associated with high seizure frequency and, when resected, showed gliosis in all six cases where...
The TSPO immunofluorescence signal indicated reactive microgliosis post injury, corresponding to PET findings. The present clinically relevant animal model and TSPO PET ligand appear to be well suited for studies on neuroinflammation after ischemic stroke....
Examination of resected cortex showed extensive neuronal death, cortical gliosis and some reactive astrocytosis in the subcortical white matter, but no specific pathological diagnosis was apparent. The general cortical architecture was preserved and the myelin pattern of the cortex and underlying white ...
Williams AJ, Hale SL, Moffett JR, Dave JR, Elliott PJ, Adams J, et al. Delayed treatment with MLN519 reduces infarction and associated neurologic deficit caused by focal ischemic brain injury in rats via antiinflammatory mechanisms involving nuclear factor-κB activation, gliosis, and leukocyte ...